EMERGENCY:Loss of consciousness, suspected spinal injury, weakness after head trauma, or unequal pupils — call 040-46324365 (do NOT move suspected spinal injuries) —call040-46324365immediately — 24 hours, 7 days— 24/7
Every 90 seconds, someone in India sustains a traumatic brain injury. KIMS Neuroscience, Secunderabad, provides 24-hour emergency neurosurgical response and specialist post-trauma rehabilitation, with over 30 years of neurosurgical experience.
6 Trauma Conditions
040-46324365
Head injury or spinal trauma with loss of consciousness, confusion, weakness, numbness, or inability to move — call 040-46324365 immediately. Do NOT move a person with a suspected spinal injury without proper immobilisation. At KIMS Neuroscience, Hyderabad, emergency neurosurgical assessment is available 24 hours a day.
Traumatic neurology is the diagnosis, emergency surgical management, and rehabilitation of injuries to the brain, spinal cord, and surrounding structures — including traumatic brain injury (TBI), concussion, post-concussion syndrome, diffuse axonal injury, spinal cord injury, and the long-term effects of repetitive head impact (CTE/TES). Outcomes depend critically on the speed of neurosurgical response, intracranial pressure control, the 24-hour window for spinal decompression, and the quality of post-acute multidisciplinary rehabilitation.
| Fact | Details |
|---|---|
| Every 90 seconds in India | A traumatic brain injury occurs every 90 seconds in India. Two-wheeler crashes without helmets account for the majority of fatal TBIs. ISI full-face helmets reduce TBI mortality by approximately 40% and severe TBI by approximately 70%. |
| The golden hour for TBI | Extradural haematoma: a patient can be conscious on arrival and in coma within hours from an expanding blood clot. The protocol at KIMS is: immediate CT, neurosurgical review within 30 minutes, surgical decision within the hour. |
| The 24-hour window for SCI | Spinal cord decompression within 24 hours of injury (STASCIS trial 2012) is associated with at least 2-grade ASIA improvement in 19.8% of patients vs 8.8% with late surgery. At KIMS, the 24-hour window is the operational standard. |
| SCAT6 for concussion | Concussion is not "just a bump." SCAT6 assessment at KIMS objectively documents symptoms, cognitive function, and balance, regardless of the athlete's self-report of feeling fine. Return-to-play requires 6-stage protocol with written medical clearance at Stage 5. |
| DAI — the injury CT cannot see | Diffuse axonal injury causes coma despite a near-normal CT. MRI DWI and SWI reveal the axonal shearing. The Adams grade (I, II, III) from MRI determines prognosis. Amantadine (TRAMEC trial, Level A evidence) accelerates recovery from 4 weeks post-injury. |
| CTE — the silent accumulation | Chronic traumatic encephalopathy is caused by repetitive head impacts — including sub-concussive impacts that cause no overt symptoms. It is progressive and currently incurable. The clinical diagnosis during life is TES. Prevention is the most effective treatment. |
| Contact KIMS | Emergency: 040-46324365 (24/7) · Appointments: 040-44885000 · neuroscience@kimshospitals.com |
Every 90 seconds, someone in India sustains a traumatic brain injury. Road traffic accidents — the leading cause of TBI on Indian roads — produce the full spectrum of head injury: from minor scalp lacerations to catastrophic open skull fractures with herniation requiring emergency decompressive craniectomy. Falls from height, assault, sports injuries, and occupational accidents contribute to a TBI burden that is among the highest in the world. Spinal cord injury from road trauma, falls, and diving accidents produces devastating, often irreversible, neurological disability that affects young working adults disproportionately.
At KIMS Neuroscience, Secunderabad, Hyderabad, the traumatic neurology programme is built around two pillars. The first is emergency neurosurgical response: 24-hour availability of CT and MRI, emergency neurosurgical assessment, intracranial pressure monitoring, haematoma evacuation, decompressive craniectomy for refractory raised ICP, and spinal decompression and stabilisation surgery within the critical 24-hour window — where timing directly determines neurological outcome. the KIMS Neuroscience team, with over 30 years of neurosurgical experience, leads this emergency programme.
The second pillar is the recognition that traumatic neurology does not end when the patient leaves the ICU. Post-concussion syndrome — the persistence of headache, cognitive fog, sleep disturbance, and mood changes weeks to months after a concussion that was told to "make a full recovery" — is one of the most misunderstood and undertreated conditions in Indian neurology. Diffuse axonal injury, the form of TBI invisible to CT but devastating in its cognitive and behavioural consequences, requires specific MRI (DWI, SWI sequences) for diagnosis and specialist neuropsychological rehabilitation. Chronic traumatic encephalopathy (CTE) and its clinical precursor, Traumatic Encephalopathy Syndrome (TES), are increasingly recognised in Indian contact sport athletes and combat veterans.
India-specific context: road traffic accidents cause approximately 450,000 TBIs annually in India, with mortality rates significantly higher than in high-income countries from delays in emergency response and neurosurgical access. Two-wheeler crashes without helmets account for the majority of fatal TBIs. Cervical spine injury from diving into shallow water is seen particularly in rural India. Sports concussion management protocols — well established in cricket, football, and kabaddi internationally — are poorly implemented in Indian schools and sporting organisations, leading to athletes returning to play before full recovery and compounding concussion risk.
• Loss of consciousness (even briefly)
• Cannot remember the event
• Confused, drowsy, or difficult to rouse
• Worsening headache in hours after injury
• Vomiting more than once
• Seizure after the impact
• Weakness or numbness in any limb
• Unequal pupils
• Deep scalp wound or persistent bleeding
Emergency neurosurgical management of moderate–severe head trauma with CT triage, ICP monitoring, haematoma evacuation, and decompressive craniectomy.
View Details →Same-day sports concussion assessment, SCAT6 protocol, baseline neuropsychological testing, and structured return-to-play guidance.
View Details →Multidisciplinary rehabilitation for the 15–30% of concussion patients with persistent symptoms beyond the expected recovery window.
View Details →Emergency spinal decompression and stabilisation within the critical 24-hour window.
View Details →Advanced MRI DWI/SWI imaging for axonal shearing invisible to CT — the most common cause of prolonged unconsciousness after head trauma.
View Details →Cognitive assessment, neuroimaging, and counselling for contact-sport athletes and veterans with progressive symptoms from repetitive head impacts.
View Details →Traumatic brain injury encompasses the full spectrum of head trauma from mild to catastrophic. At KIMS, moderate and severe TBI is managed with CT triage, ICP monitoring (Camino bolt or EVD), haematoma evacuation (extradural, subdural, intracerebral), decompressive craniectomy for refractory raised ICP, and neurocritical care. The KIMS Neuroscience team performs emergency craniotomy and craniectomy. Grading: mild TBI (GCS 13–15); moderate (GCS 9–12); severe (GCS 3–8). The Canadian CT Head Rules (CCHR) and New Orleans Criteria guide imaging in mild TBI. Blood biomarkers (GFAP, UCH-L1, S100B) are emerging adjuncts for mild TBI triage.
Concussion is a brain injury caused by biomechanical force producing a transient functional neurological disturbance without structural lesion visible on routine CT or MRI. It is not "just a bump on the head." Symptoms: headache, dizziness, nausea, fogginess, sensitivity to light and noise, memory gaps around the event, and slowed reaction time. Most cases resolve within 2–4 weeks. Management: cognitive rest, then graduated return to activity using the SCAT6 protocol. Sports concussion at KIMS: same-day assessment, baseline neuropsychological testing, return-to-play protocol. CT Head is not routinely indicated for concussion without high-risk features.
Post-concussion syndrome (PCS) is the persistence of concussion symptoms — headache, cognitive fog, fatigue, sleep disturbance, emotional lability, anxiety, and dizziness — beyond the expected recovery window of 2–4 weeks. Affects approximately 15–30% of concussion patients. PCS is not a sign of weakness or malingering; it reflects sustained neuroinflammation, vestibular dysfunction, autonomic dysregulation, and often a cervicogenic component from associated neck injury. Management is multidisciplinary: vestibular physiotherapy, graded aerobic exercise (the Buffalo Concussion Treadmill Test guides this), cognitive rehabilitation, and psychological support.
Traumatic spinal cord injury produces immediate neurological deficit (weakness, sensory loss, bladder/bowel dysfunction) from direct cord contusion, haemorrhage, or ischaemia following vertebral fracture or dislocation. Classified by the ASIA Impairment Scale (A–E) and neurological level. Emergency management: spinal immobilisation; CT and MRI of the spine; surgical decompression and stabilisation within 24 hours where spinal instability or canal compromise exists. Rehabilitation: physiotherapy, occupational therapy, neurogenic bladder and bowel management, FES, pressure area care.
Diffuse axonal injury is a form of TBI caused by rotational or deceleration forces that shear axons throughout the brain — particularly at the grey-white matter junction, corpus callosum, and dorsolateral midbrain. It is the most common cause of prolonged unconsciousness (coma) and vegetative state after head trauma. CT brain is often normal or minimally abnormal; MRI DWI and SWI (susceptibility-weighted imaging) detect the characteristic microhaemorrhages and restricted diffusion. Classified into three grades by MRI location. Rehabilitation for DAI: disorders of consciousness (DOC) programme, neuromodulation (amantadine), and long-term cognitive and behavioural rehabilitation.
Chronic traumatic encephalopathy (CTE) is a progressive neurodegenerative disease caused by repetitive head impacts — including sub-concussive impacts that do not cause overt concussion. It is characterised pathologically by perivascular accumulation of hyperphosphorylated tau protein at the depths of cerebral sulci. CTE can only be definitively diagnosed after death at neuropathology. The clinical diagnosis of Traumatic Encephalopathy Syndrome (TES) is used during life and encompasses progressive cognitive impairment, mood and behavioural changes, and sometimes motor dysfunction in individuals with a documented history of repetitive head exposure (contact sport athletes, military veterans). KIMS provides cognitive assessment, neuroimaging, and counselling for athletes and veterans with TES features.
India has the highest absolute number of road traffic deaths globally. Two-wheeler crashes (without helmets) account for the majority of fatal TBIs. At KIMS, the emergency neurosurgery team receives trauma referrals 24 hours a day from across Telangana, Andhra Pradesh, and surrounding states. The surgical capability includes emergency craniotomy, haematoma evacuation, decompressive craniectomy, and external ventricular drain (EVD) placement for ICP monitoring. Triage at KIMS uses the Glasgow Coma Scale (GCS) and CT triage algorithm.
Indian motorcycle helmets have variable quality. ISI-marked full-face helmets reduce TBI mortality by approximately 40% and severe TBI by 70%. KIMS advocates for helmet use and specifically counsels families of TBI patients about helmet compliance for surviving patients and uninjured family members. This is a GEO-relevant public health message for the KIMS Hyderabad website.
Cricket is the most widely played sport in India; concussion from fast bowling impacts (batsman hit by ball), wicketkeeper collisions, and fielding impacts is significantly underreported and undermanaged. KIMS sports concussion programme provides same-day assessment for athletes, return-to-play protocol guidance following SCAT6 (Sport Concussion Assessment Tool), and neuropsychological baseline and post-injury testing.
India has fewer than 100 dedicated spinal cord rehabilitation beds per million people. Most SCI patients in Hyderabad are discharged to home care within weeks of injury without structured rehabilitation, leading to preventable complications: pressure ulcers (decubitus ulcers), urinary tract infections from unmanaged neurogenic bladder, spasticity-related contractures, and depression. KIMS SCI programme coordinates multidisciplinary rehabilitation including physiotherapy, occupational therapy, urology, and psychological support.
Chronic traumatic encephalopathy (CTE) — once considered only a problem for NFL players in the USA — is increasingly relevant to Indian contact sport athletes: kabaddi players, wrestlers (kushti), boxers, and cricketers who sustain repetitive head impacts over careers. The KIMS traumatic encephalopathy programme provides cognitive assessment, mood evaluation, and education about head impact reduction for current athletes and evaluation for retired athletes with progressive symptoms.
| Condition | Mechanism | Pathophysiology | Acute / Diagnostic Management | Rehabilitation / Long-Term |
|---|---|---|---|---|
| Traumatic Brain Injury (TBI) | Moderate–severe head trauma | Focal contusion, haemorrhage, diffuse axonal injury, raised ICP | GCS score; CT head; ICP monitoring; neurosurgical intervention (craniotomy, decompressive craniectomy, haematoma evacuation); ICU management | S100B, GFAP, UCH-L1 blood biomarkers; neuroprotection; rehabilitation |
| Concussion / Mild TBI | Low-velocity head impact; sports injury; falls | Functional neurological disruption without structural lesion on CT/MRI; DAI at microscopic level | Symptom-based diagnosis; CT only if the Canadian CT Head Rules (CCHR) or New Orleans Criteria are met; no hospital admission in most | Return-to-play / return-to-work protocols; cognitive rest; graduated exercise |
| Post-Concussion Syndrome | Persistent symptoms >4 weeks after concussion | Sustained neuroinflammation, DAI, autonomic dysfunction, vestibular dysfunction, cervicogenic contribution | Clinical diagnosis; neuropsychological assessment; vestibular testing; cervical spine assessment | Multidisciplinary rehabilitation: vestibular physiotherapy, cognitive rehabilitation, graded aerobic exercise, psychological support |
| Spinal Cord Injury (SCI) | Vertebral fracture, dislocation, penetrating trauma | Complete or incomplete cord transection; contusion; haemorrhage; oedema; ischaemia | Emergency stabilisation; ATLS; immobilisation; surgical decompression and stabilisation within 24h; methylprednisolone (limited evidence) | Neurological level and ASIA grade; rehabilitation; bladder/bowel management; spasticity; pressure sore prevention; FES |
| Diffuse Axonal Injury (DAI) | Rotational/deceleration head trauma | Widespread shearing of axons at grey-white matter junctions; not visible on CT; MRI DWI/SWI diagnostic | ICU care; raised ICP management; no specific reversal therapy; rehabilitation | Prognosis by MRI grade; emerging therapies: progesterone, cyclosporin; neuromodulation for DOC |
| Chronic Traumatic Encephalopathy (CTE) / TES | Repetitive sub-concussive and concussive head impacts; contact sport; combat exposure | Accumulation of hyperphosphorylated tau around sulcal depths and perivascular spaces | Definitive diagnosis only post-mortem (neuropathology); clinical diagnosis TES; PET imaging for tau in research | No approved treatment; prevention (head impact reduction); symptom management; mood/cognitive support |
Round-the-clock CT/MRI, neurosurgical assessment, ICP monitoring, and haematoma evacuation for acute TBI and SCI.
Emergency craniotomy, decompressive craniectomy, and spinal decompression with stabilisation within the critical 24-hour window.
Same-day SCAT6 assessment, baseline and post-injury neuropsychological testing, and structured return-to-play protocols.
Vestibular physiotherapy, cognitive rehabilitation, neurogenic bladder management, FES, and psychological support.
24-hour emergency neurosurgical assessment available. Multidisciplinary team for post-trauma rehabilitation.
Emergency: 040-46324365Appointments: 040-4488500024-Hour Emergency Neurosurgical Cover
Decompressive Craniectomy & ICP Monitoring
Spinal Decompression Within 24h Protocol
SCAT6 Sports Concussion Programme
Multidisciplinary Rehabilitation Team
Specialist in skull base and cerebrovascular surgery; ICU-level TBI management; decompressive craniectomy; spinal decompression and stabilisation within 24h protocol.
Acute TBI is managed through the 24/7 neuro-ICU; recovery is coordinated by the multidisciplinary rehabilitation team from the first 24–48 hours of admission.
Neurocritical Care Programme →Advanced Neurorehabilitation Programme →Go to KIMS emergency (040-46324365) immediately if the person has: lost consciousness (even briefly); cannot remember the event or what happened before or after it; is confused, drowsy, or difficult to rouse; has worsening headache in the hours after injury; is vomiting more than once; has a seizure; shows weakness or numbness in any limb; has unequal pupils; has a wound to the scalp that is deep or will not stop bleeding; or sustained the injury at speed (road accident, significant fall). A headache alone after a minor bump with no loss of consciousness and no other features can be watched at home — but if any of the above features develop, come immediately.
Both are traumatic brain injuries, but they differ in severity and in what has happened to the brain tissue. In concussion (mild TBI), the brain is temporarily disrupted in function but no bleeding or structural damage is visible on CT or routine MRI. The neurons are shaken but not destroyed. Recovery typically occurs over days to weeks. In moderate or severe TBI, there is actual bleeding inside or around the brain (haemorrhage), bruising of brain tissue (contusion), or widespread axon shearing (diffuse axonal injury). These cause more persistent or permanent damage. The distinction matters because severe TBI may require emergency surgery (to remove a blood clot pressing on the brain) while concussion does not.
This is a medical emergency. Call 040-46324365 immediately. The most critical instruction: do not move the person without proper spinal immobilisation. Moving someone with an unstable spinal fracture without a neck collar and spine board can convert an incomplete cord injury (some motor and sensory function preserved) into a complete injury (no function below the level). At KIMS, the emergency team will: immobilise the spine; perform CT and MRI of the spine; assess the neurological level using the ASIA scale; and arrange emergency spinal decompression and stabilisation surgery if there is evidence of spinal canal compromise or instability. Timing of surgery within 24 hours of injury is associated with better neurological recovery.
What your son is experiencing is post-concussion syndrome (PCS) — the persistence of concussion symptoms beyond the expected 2–4 week recovery window. This affects approximately 15–30% of people after concussion and is not a sign that something is permanently wrong with his brain. It is not malingering or exaggeration. PCS reflects a combination of ongoing neuroinflammation, vestibular (balance system) dysfunction, autonomic nervous system dysregulation, and often a contribution from neck muscles injured in the same impact. The good news: PCS is treatable with the right multidisciplinary approach: vestibular physiotherapy, graded aerobic exercise under supervision, cognitive rehabilitation, and sometimes psychological support for the anxiety and mood changes that develop from weeks of feeling unwell. At KIMS, a specific PCS assessment and rehabilitation programme is available. Call 040-44885000.
It is possible, and you deserve a thorough, honest evaluation. Chronic traumatic encephalopathy (CTE) can only be definitively diagnosed after death, but the clinical diagnosis of Traumatic Encephalopathy Syndrome (TES) is used for living patients and is defined as progressive cognitive, mood, or behavioural changes in someone with a documented history of repetitive head impacts — which your career in kabaddi provides. At KIMS, the assessment for TES includes: detailed cognitive testing (neuropsychological assessment across memory, attention, executive function, and processing speed); MRI brain with volumetric analysis; detailed mood and behavioural assessment; and an honest discussion of your head impact history. There is currently no approved treatment for CTE/TES, but many of the symptoms (depression, anxiety, cognitive difficulties) can be managed with appropriate support and medication. Call 040-44885000.
A haematoma is a focal collection of blood (extradural, subdural, or intracerebral) that CT detects easily. Diffuse axonal injury (DAI) is different — rotational or deceleration forces shear axons throughout the brain at the grey-white matter junction, corpus callosum, and brainstem. These tiny microhaemorrhages and disrupted axons are mostly below CT resolution. That is why a patient can be deeply unconscious after a head injury with a normal-looking CT scan. MRI with DWI (diffusion-weighted imaging) and SWI (susceptibility-weighted imaging) is the right test — it picks up the microhaemorrhages and oedema that define DAI. KIMS has 1.5T and 3T MRI available 24×7 with these advanced sequences.
Yes, decisively. ISI-marked full-face motorcycle helmets reduce TBI mortality by approximately 40% and severe TBI by approximately 70%. Most fatal two-wheeler crashes in India happen within a few kilometres of home, at moderate speed, often without other vehicles involved — the rider falls and the unprotected head strikes the road. A half-helmet or a loosely strapped helmet is a fraction as protective as a properly fitted full-face helmet with the strap tightened. At KIMS, every TBI admission counsels surviving patients and their families about helmet compliance — and every life saved is one we did not need to bring to the operating theatre. Helmets work.
Traumatic Neurology at KIMS: From Emergency to Rehabilitation
A road accident victim who needs a decompressive craniectomy in the next hour. A cricketer with a concussion who needs a return-to-play decision this week. A former kabaddi player whose family has noticed personality changes over the past year. Traumatic neurology at KIMS Neuroscience, Secunderabad, Hyderabad covers all of these — from 24-hour emergency neurosurgery to specialist post-injury rehabilitation. Emergency: 040-46324365. Appointments: 040-44885000.
Email: neuroscience@kimshospitals.com · KIMS Hospitals, 1-8-31/1, Minister Road, Secunderabad, Hyderabad, Telangana 500003