EMERGENCY:Sudden severe vertigo with headache, double vision, slurred speech, weakness, or facial asymmetry (possible posterior-fossa stroke) —call040-46324365immediately — 24 hours, 7 days— 24/7
Expert diagnosis and treatment of vestibular and balance disorders at KIMS Hyderabad — BPPV, vestibular neuritis, Menière's disease, central vertigo, POTS, and orthostatic hypotension.
6 Major Disorders
040-44885000
VNG · HINTS · Tilt Table
040-46324365
Vestibular and balance disorders are conditions of the vestibular system — the inner ear, vestibular nerve, brainstem, and cerebellum — that produce vertigo (an illusion of movement), dizziness, imbalance, or near-fainting. They range from the very common and very treatable (BPPV, vestibular neuritis) to neurological emergencies (posterior-fossa stroke presenting as vertigo). The most useful early distinction is peripheral (inner-ear or vestibular-nerve) vs central (brainstem or cerebellar) — the HINTS bedside exam at KIMS makes this distinction faster and more reliably than early MRI. KIMS Neuroscience Hyderabad treats: BPPV · Vestibular Neuritis · Menière's Disease · Central Vertigo · POTS · Orthostatic Hypotension.
| Fact | Details |
|---|---|
| Conditions covered | BPPV · Vestibular Neuritis · Menière's Disease · Central Vertigo · POTS · Orthostatic Hypotension |
| BPPV in plain terms | Brief (<1 min) spinning triggered by lying down, rolling, or looking up. Affects ~2.4% of adults over a lifetime. Epley manoeuvre cures >90% of cases in 1–3 visits |
| Vestibular neuritis | Sudden severe continuous vertigo lasting days · No hearing loss · Steroids accelerate recovery · Vestibular rehabilitation is the cornerstone of long-term recovery |
| Menière's disease tetrad | Episodic vertigo (20 min–2 h) + Fluctuating hearing loss + Tinnitus + Aural fullness — all in the same episode |
| HINTS exam | Head Impulse + Nystagmus + Test of Skew · Bedside · Distinguishes peripheral vertigo from posterior-fossa stroke · More sensitive than MRI in the first 24h for posterior fossa stroke |
| POTS vs OH | POTS: heart rate rises ≥30 bpm on standing, BP often preserved · OH: BP drops ≥20 mmHg systolic on standing · Both diagnosed at KIMS by tilt table test |
| Contact | 040-44885000 (outpatient) · 040-46324365 (emergency, 24/7) · No referral required |
Dizziness is one of the most common complaints in medicine — affecting an estimated 15–25% of adults at some point in their lives and accounting for approximately 4% of all emergency department visits globally. Yet "dizziness" is a word that patients use to describe very different experiences: the spinning sensation of true vertigo, the lightheadedness of a blood pressure drop on standing, the floating unsteadiness of chronic balance impairment, and the near-fainting of a vasovagal episode.
The vestibular system is the brain's mechanism for detecting head position and movement in space. It consists of the inner ear labyrinth (the three semicircular canals detecting rotational acceleration in three planes, and the utricle and saccule detecting linear acceleration and gravity) and the central vestibular pathways in the brainstem and cerebellum. Disorders at any level of this system produce dizziness and balance impairment.
• New-onset severe vertigo with headache
• Double vision
• Slurred speech
• Limb weakness
• Inability to walk
• Facial asymmetry
Distinguishing between different sources of dizziness — and identifying the correct anatomical source — is the defining challenge of vestibular medicine.
Intense spinning, nausea, positional, fatigable, horizontal nystagmus with fast phase away from lesion, preserved gaze fixation, no focal neurological signs.
May be mild, direction-changing nystagmus, vertical nystagmus, HINTS exam positive, focal signs, skew deviation.
Head Impulse (normal in central) + Nystagmus (direction-changing or vertical = central) + Test of Skew (skew deviation = central). Performed at the bedside to distinguish central from peripheral vertigo — more sensitive than MRI in the first 24 hours for posterior fossa stroke.
The most common cause of vertigo worldwide. Displaced otoconia from the utricle enter a semicircular canal.
View Details →Acute unilateral vestibular deafferentation from viral inflammation of the vestibular nerve.
View Details →Idiopathic endolymphatic hydrops — accumulation of excess endolymph in the membranous labyrinth.
View Details →Vertigo from brainstem or cerebellar pathology. A neurological emergency when caused by stroke.
View Details →Dizziness, lightheadedness, and near-fainting on standing from a heart rate surge rather than true vestibular vertigo.
View Details →Fall in systolic BP ≥20 mmHg on standing causing dizziness, greying of vision, and near-fainting.
View Details →Videonystagmography (VNG), Dix-Hallpike and Roll tests, Head Impulse Test (video-HIT), Caloric testing, and VEMP.
Epley, Semont, and BBQ roll canalith repositioning — highly effective (>90% resolution) for BPPV.
Head Impulse + Nystagmus + Test of Skew — more sensitive than MRI in the first 24h for posterior fossa stroke.
VRT, gaze stabilisation exercises, balance retraining, and falls prevention programme.
BPPV: most common, responds to Epley manoeuvre
Diabetes + hypertension: common causes of vestibular neuropathy and OH in India
Ototoxic medications (aminoglycoside antibiotics) widely used in India
Central vertigo: brainstem strokes from vertebrobasilar disease
Hyderabad summer heat: worsens OH and vasovagal episodes
Our vestibular neurology and otoneurology teams review every complex case. No referral required.
Videonystagmography (VNG) & Video-HIT
Bedside HINTS Examination
Tilt Table Testing for POTS & OH
Vestibular Rehabilitation Therapy (VRT)
This is the classic description of Benign Paroxysmal Positional Vertigo (BPPV) — by far the most common cause of vertigo, affecting approximately 2.4% of adults over a lifetime. BPPV is caused by tiny calcium carbonate crystals (otoconia) from the utricle in the inner ear dislodging and migrating into one of the semicircular canals. When you move your head in the plane of the affected canal, these crystals move and trigger a false signal of rotation, producing the brief spinning sensation. The key features that confirm BPPV: the vertigo is brief (usually 10–30 seconds, always less than a minute); it is triggered by specific head positions (lying down, rolling to one side, looking up); and it is not associated with hearing loss or focal neurological symptoms. A simple diagnostic test called the Dix-Hallpike manoeuvre at KIMS confirms the diagnosis in minutes. The treatment — the Epley repositioning manoeuvre — is performed in the clinic and resolves BPPV in over 90% of patients after 1–3 treatments. Call 040-44885000.
The picture you describe — severe continuous vertigo lasting many hours with vomiting and inability to walk, but without headache and without hearing loss — is most consistent with vestibular neuritis (inflammation of the vestibular nerve), not a stroke. Vestibular neuritis is caused by viral inflammation of the vestibular nerve and produces acute, severe, continuous (not positional) vertigo lasting hours to days. The absence of headache, double vision, slurred speech, limb weakness, or facial asymmetry argues against a posterior fossa stroke. However, the HINTS examination at KIMS (a bedside test of eye movements) is the most important step to distinguish vestibular neuritis (peripheral) from a brainstem or cerebellar stroke (central). If there is ANY suspicion of central pathology, an MRI brain with DWI is performed immediately. At KIMS, the emergency neurology team performs the HINTS exam in all patients with acute vertigo and selects MRI when needed. Call 040-44885000, or 040-46324365 if symptoms are severe.
The symptom cluster you describe — episodic vertigo lasting 20 minutes to 2 hours, with fullness in one ear, tinnitus (roaring sound), and fluctuating hearing loss all occurring together in the same episode — is the classic tetrad of Menière's disease. Menière's is caused by excess endolymph (inner ear fluid) distending the membranous labyrinth — a condition called endolymphatic hydrops. The distension affects both the cochlea (causing hearing loss and tinnitus) and the vestibule (causing vertigo), which is why all four symptoms occur together. Between attacks, the patient often feels completely normal initially, but over years the hearing loss becomes progressively permanent. The diagnosis at KIMS is confirmed by audiometry (low-frequency sensorineural hearing loss) and electrocochleography (ECochG). Treatment begins with dietary salt restriction and diuretics to reduce endolymph pressure. Call 040-44885000.
No — a CT scan is not sufficient to exclude a posterior fossa (brainstem and cerebellum) stroke. CT is very insensitive for acute ischaemic strokes in the posterior fossa — the dense bone of the posterior skull obscures the brainstem and cerebellum on CT, and small infarcts in these critical structures can be invisible on CT even 24–48 hours after onset. MRI with diffusion-weighted imaging (DWI) is the gold standard for detecting acute posterior fossa infarction. A normal DWI in the first 24–48 hours does not completely exclude an acute brainstem infarct (early DWI sensitivity is approximately 80% for acute posterior fossa ischaemia), which is why the clinical examination — particularly the HINTS exam — is critical. The HINTS exam (assessing head impulse, nystagmus direction, and skew deviation) has been shown to be more sensitive than early MRI for posterior fossa stroke in acute vertigo. If the HINTS exam raises concern for a central cause, MRI should be repeated at 48–72 hours even if the first MRI is negative. Call 040-44885000.
Yes, the distinction is the most useful first question in a balance clinic. Vertigo is an illusion of movement — usually spinning — that strongly suggests a vestibular cause (BPPV, vestibular neuritis, Menière's, or central vertigo). Dizziness without spinning is more often non-vestibular: orthostatic hypotension (lightheadedness on standing), POTS (palpitations + lightheadedness on standing), cardiac (rhythm or perfusion problems), or psychogenic. The history and the tilt table or HINTS exam at KIMS sort out which kind.
Yes — direct vestibular and balance-clinic appointments are accepted. Outpatient: 040-44885000 (Mon–Sat). For sudden severe vertigo with headache, double vision, slurred speech, limb weakness, or facial asymmetry, call 040-46324365 immediately — these features can indicate a posterior-fossa stroke.
Initial consultation is 30–60 minutes. The Dix-Hallpike test and Epley manoeuvre for BPPV are usually performed at the same visit. Extended testing (VNG, vHIT, caloric, VEMP, tilt table) is usually scheduled in a single half-day block. A complete diagnostic picture and a rehabilitation plan are typically in place within 1–2 weeks.
One Centre for All Vestibular and Balance Care
The world spinning every time you turn over in bed. A sudden severe vertigo that came out of nowhere. Hearing and balance failing together in one ear. Lightheadedness every time you stand up. At KIMS Hyderabad, the vestibular neurology and otoneurology teams provide the Dix-Hallpike test, the Epley manoeuvre, VNG, HINTS exam, tilt table testing, and vestibular rehabilitation — all in one centre.
Emergency: 040-46324365 · Outpatient: 040-44885000